Ka'aʻaiawāʻana o ka'ōpū: etiology a me ka pathogenesis

Ka'aʻaiawāʻana o ka'ōpū - he 'ano, he pono ole maʻi, ua wehewehe aku ma ke kahua o erosions ma luna o ka ili o ka mucousʻili lahilahi o ka'ōpū. Mau mā hou no ka mea, o ka superficial necrosis. I kā lākou ho'ōla connective me ka makaʻu no uaʻaʻole i hanaʻia. Ka'aʻaiawāʻana o ka'ōpū - mea he conditional nosological kau mea hiehie recurrent maʻi luku keʻano o ka hanaʻana i loko o ka gastric mucosa. I kekahi manawa e hoike mai ai ia ia iho me he symptom o gastritis. No laila, i loko o ka moʻokalaleo pinepine ua kapaia keia maʻi erosive gastritis.

I ka papa kuhikuhiE kumu o ka maʻi i loa ? Aneie aʻe i i mau hana ma luna o ke kanaka kino. Mau stress kumumea ('ā, frostbite, poino, kŰia, polytrauma) i ka 'aʻe' ana o neurohumoral maomeka o ka hooponopono ana o na mea he nui nā loko, pakahi aku la ia i kaʻai a me ka secretory oihana o ka opu a me ka naʻau liʻiliʻi, emi ka ia'ku o ka meaola. Aia Ua hoi he aaiaoe predisposition i ka maʻi. Ka'aʻaiawāʻana o ka'ōpū i kekahi e ala aʻeʻoe i loko o ka hoʻohanaʻana i lapaʻauʻia (nitrofurans, corticosteroids, ethacrynicʻakika, veroshpiron, tolbutamide), ka waiʻona hainā, e like me nā maʻi o ka endocrine'ōnaehana (mimikō, hyperparathyroidism).

He nui nā manaʻo i ka erosive gastritis kupu mai ka pathogenic hana o ka Campylobacter (Helicobacter pylori) i loko o ke kanaka kino. Kūʻiʻo Ua hoike ia 95% o nā poʻe me ka duodenal ulcers i iho mailoko me like keia ano o Campylobacter, akā, nā mea maʻi me ka mau ulcers hoike no wale 1-6% o nā kanaka mailoko o me ka pathogen.

Ka'aʻaiawāʻana o ka'ōpū: pathogenesis (maʻi Holomua)

Ma lalo o ka aoao o ka stress kumumea i loko o ke kino aia no i luna 'a me ka i waena o-pili biochemical keʻano o ka hanaʻana, a komo pū' e neural a me ka humoral maomeka o ka rula hooponopono o ka mana o nā mea a pau kino nenoai, ka oi aku o ka'ōpū a me ka naʻau liʻiliʻi. Hoonuiia secretion o adrenal cortex hormones a me histamine, i ka hōʻoi 'ka secretion o gastric wai me ka hoomahuahua i ka hope o ka pepsin a me kona maʻiʻo o noa hydrochloricʻakika. Ka poʻe ua hoʻemi (viscosity) o ka mucus secretion, a i ho'ēmi i kā aiiay mucosal kuleana pili i. Eia kekahi, he spasmodic contraction o nāʻiʻo o ka opu a me ka naʻau liʻiliʻi, mahuahua capillary permeability,'aʻaʻa trophism nee, e hiki aku ai a laila i ka hoʻoʻino reactions, ho okumu ana o erosions a me ulcers o ka gastric mucosa. It E e mahalo a me ka koke hopena o nā palapalaʻawahia waiwai i ka mucosa. Ke'ī mai, ma ka pathogenesis o gastric ka'aʻaiawā mea he ki hehee ai o ka discrepancy ma waena o ka papa mucosal ke'ākeʻakeʻa mai, a me ka lumai hiki o gastric a intestinal Contents. Haawi mai i ka helu ana o na moku i ke koko i loko o ka wahi hana kahe ana ke koko ulcer. Ma keia maʻi hoʻomōhala ku paa auanei ooieoeiiaeuiiai opu kāna hana, liʻiliʻi, ake, pancreas a me nā nā loko, a nenoai.

More pinepine (30-90%) i loko o ui kanaka i ka maʻi 'Aʻole maopopo hoailona. I kekahi manawa hoike mai ai ia heartburn,ʻakika regurgitation, kākaʻikahi ilaila e e eha i loko o ka'ōpū. Maʻi erosive gastritis hoike mai ai ia me ka typical lapaʻau hoailona. About 80% o nā mea maʻi ohumu o heartburn, belching,ʻeha ma ka akau maluna quadrant, bloating, diarrhea a me ka constipation.

O ka poe i kauka diagnosed erosive gastritis, e pono e hahai i kaʻai nona ma hoemi gastric haʻawina. E loaʻa ai i kēia pahuhopu e pono eʻai'ākau. Mua o nā mea a pau, iʻai no gastric ka'aʻaiawā komo ai i ka hoole ana mai a lohi puluniuʻai i ka mea waiwai ma ka kelulose (radishes, stringy ai, turnips, bran berena, etc.). Oe hiki ole ai Pōʻalima ai. Ka mea, e hookaaokoa lakou i na kanakaʻona mau nā mea inu, carbonated ka wai, citrus, kope,ʻeleʻele i ka berena, e like me ka pono me ke anu a me ka wela ai.